 |
Journal articles
Chemotherapy-elicited upregulation of NKG2D and DNAM-1 ligands as a therapeutic target in multiple myeloma.
Abstract : Malignant cells constitutively express Natural killer group 2, member D (NKG2D) or DNAX Accessory Molecule-1 (DNAM-1) ligands, yet they are often unable to trigger a robust cytotoxic cell response. It may be therapeutically useful to implement strategies aimed at increasing the density of NKG2D and DNAM-1 ligands on the surface of cancer cells, endowing them with the capacity to activate potent antitumor natural killer-cell responses.
Cited literature [9 references]
https://hal-riip.archives-ouvertes.fr/pasteur-01054170
Contributor : Istituto Pasteur Fondazione Cenci Bolognetti Connect in order to contact the contributor
Submitted on : Tuesday, August 5, 2014 - 11:55:18 AM
Last modification on : Friday, November 25, 2022 - 7:04:09 PM
Long-term archiving on: : Tuesday, April 11, 2017 - 6:57:09 PM
Contributor : Istituto Pasteur Fondazione Cenci Bolognetti Connect in order to contact the contributor
Submitted on : Tuesday, August 5, 2014 - 11:55:18 AM
Last modification on : Friday, November 25, 2022 - 7:04:09 PM
Long-term archiving on: : Tuesday, April 11, 2017 - 6:57:09 PM
File
Soriani_et_al.pdf
Publication funded by an institution